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- Commensal Cutibacterium acnes-derived indolelactic acid safeguards skin barrier function through the aryl hydrocarbon receptor.
- Skin Barrier Dysfunction, Antimicrobial Peptide Alterations, and Microbiome Changes in Solid Cancer Patients Treated with Epidermal Growth Factor Receptor Inhibitors.
- Enhancement of Skin Barrier Function Using DYNAMiQ Technology: A Clinical Evaluation.
Clinical Evidence on Water
Commensal Cutibacterium acnes-derived indolelactic acid safeguards skin barrier function through the aryl hydrocarbon receptor.
This study highlights the role of indolelactic acid from Cutibacterium acnes in restoring skin barrier integrity and mitigating dermatitis. Read more.
Skin Barrier Dysfunction, Antimicrobial Peptide Alterations, and Microbiome Changes in Solid Cancer Patients Treated with Epidermal Growth Factor Receptor Inhibitors.
Research indicates that epidermal growth factor receptor inhibitors compromise skin barrier function, affecting water loss and microbiome. Read more.
Enhancement of Skin Barrier Function Using DYNAMiQ Technology: A Clinical Evaluation.
This evaluation demonstrates that DYNAMiQ technology significantly improves skin barrier function by reducing transepidermal water loss. Read more.
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